Novavax Exposed!

by Celeste Solum July 30, 2022

I presented this information in the spring of 2020 in my Webinar series, Pathos of our Great Delusion at Celetialreport.com.

Bottom line Norovax is a soul catcher. Please join me beginning this Tuesday for a 6-part Soul Catcher Webinar with written articles describing in graphic detail what these gene editing weapons does to your spiritual life through the use of technology and witchcraft.

What is the difference between the soul, animal soul, and life?
Is the soul borrowed from God?
Where is the soul located in the body?
Is it possible for you to be stripped of your soul from your body becoming a disembodied soul?
Does a hybrid have a soul?
Where does the hybrid go upon death- cessation of life, or do they even have life?
Is it possible for a disembodied soul to inhabit a Synth (synthetic body)?
Will a disembodied soul go to heaven?

Bottom Line: If you take this gene editing weapon you become are a victim of Direct and Experimental Evolution that changes you from a human to a hybrid.

Basic Facts

DHS: Novavax has begun a Phase III trial of its subunit vaccine candidate NVX-CoV2373.

Name: NVX-CoV2373 vaccine

Country: United States

Manufacturer’s: Novavax

Dose: Unknown

Vaccine Type: Recombinant nanoparticle technology with patented saponin-based Matrix-M™ adjuvant, Subunit, Baculovirus

Ingredient List: Proprietary

Professional Insert: None Provided

Dose: 2-dose

Ideology: Our vaccine candidates are genetically engineered using three-dimensional nanostructures of recombinant proteins critical to disease pathogenesis.

Baculovirus as Vaccine Vector

This vaccine uses a baculovirus: LINK, LINK

Application of viral vectors derived from human viruses to mediate immune response in animals and humans has been greatly hampered by the problems associated with pre-existing immunity and associated toxicities. Among few non-human viral vectors, baculovirus has now evolved as a novel tool for vaccine vector development.

Baculovirus has now attained a niche position in the arena of vaccine development.

Recombinant envelope-modified [protein gp64 or other viral protein] LINK baculovirus equipped with novel shuttle promoters [promoter-selection vector containing a promoterless gene and allows the identification of promoters used in inserting or coding this into another species] for in vivo transduction has shown promising results.

Baculovirus mediated induction of systemic and mucosal immune responses through intranasal or oral administration.

PPV [Porcine parvovirus (PPV)] VLPs [Virus-like particles (VLPs) are multiprotein structures that mimic the organization and conformation of authentic native viruses but lack the viral genome] could be contaminated by recombinant baculoviruses.

Baculoviruses can be a problem for the registration and safety requirements of the recombinant vaccine, we have tested different baculovirus inactivation strategies, studying simultaneously the integrity and immunogenicity of the VLPs.

Methods of Chemical Inactivation or Neutralization of a Virus

Pasteurization,
Treatment with detergents and
Alkylation with binary ethylenimine (BEI)
Triton X-100

Binary-ethylenimine (BEI) is formed by the cyclisation of 2-haloalkylamines under alkaline conditions, which reaction can be carried out either in the virus suspension, or separately, prior to addition." It supposedly chemically inactivates or neutralizes a virus.

Binary ethylenimine and Triton X-100 inactivated particles maintained all the original structural and antigenic properties. LINK

What is a Subunit?

Assembly of the Moth Cell Nanoparticles

A subunit is a protein subunit vaccine, which means that it uses a lab-made version of the SARS-CoV-2 spike protein. Novavax has inserted an ingredient called an adjuvant, which acts as a hype man for the immune system to signal it to spring into action.

They use a bioreactor containing genetically-modified moth cells. The researchers harvested the spike proteins from the moth cells and assembled them into nanoparticles. While the nanoparticles mimicked the molecular structure of the coronavirus, they could not replicate or cause Covid-19.

In structural biology, a protein subunit is a single protein molecule that assembles (or "coassembles") with other protein molecules to form a protein complex.

Some naturally occurring proteins have a relatively small number of subunits and therefore described as oligomeric, for example hemoglobin or DNA polymerase.

Others may consist of large numbers of subunits called multimeric, microtubules and other cytoskeleton proteins.

Subunits of a multimeric protein may be identical, homologous or totally dissimilar and dedicated to disparate tasks. One subunit may be a "catalytic subunit" that enzymatically catalyzes a reaction.

A "regulatory subunit" will facilitate or inhibit the activity.

Although telomerase has telomerase reverse transcriptase as a catalytic subunit, regulation is by factors outside the protein.

[1] An enzyme composed of both regulatory and catalytic subunits when assembled is often referred to as a holoenzyme.

[2] One subunit is made of one polypeptide chain. A polypeptide chain has one gene coding for it – meaning that a protein must have one gene for each unique subunit.

A subunit is often named with a Greek or Roman letter, and the numbers of this type of subunit in a protein are indicated by a subscript. From Wikipedia

Matrix M

M™, the adjuvant component of the vaccine, in order to enhance the immune response and stimulate high levels of neutralizing antibodies. NVX-CoV2373 is a stable, prefusion protein made using Novavax' proprietary nanoparticle technology.

We are also developing proprietary immune-stimulating saponin-based adjuvants at Novavax AB, our wholly owned Swedish subsidiary. Matrix-M™ has been shown to enhance immune responses.

Matrix-M™, an adjuvant based on saponin extracted from the Quillaja saponaria Molina tree LINK induces high and long-lasting levels of broadly reacting antibodies supported by a balanced TH1 and TH2 type of response, including biologically active antibody isotypes such as murine IgG2a, multifunctional T cells and cytotoxic T lymphocytes. The mode-of-action of Matrix-M adjuvant has not been elucidated in detail; however, the adjuvant promotes rapid and profound effects on cellular drainage to local lymph nodes creating a milieu of activated cells including T cells, B cells, Natural Killer cells, neutrophils, monocytes and dendritic cells.

Matrix-M has shown potent adjuvant activities, inducing enhanced immune responses in and human clinical studies, most recently with H7N9 avian influenza virus like particle vaccine where Matrix-M adjuvanted vaccine had a significant dose-sparing effect and an acceptable safety profile

NHP protection data involve the use of viral vectors or viral chimaeras. Inactivated virus and sub-unit approaches have had limited success, particularly in non-human primates. However, there are some positive studies using potent TH1 type stimulatory adjuvants.

In the present study, we used EBOV/Mak GP nanoparticles produced in Sf9 cells infected with recombinant baculovirus expressing EBOV/Mak GP [28] combined with Matrix-M. Mice were immunized with EBOV/Mak GP alone or adjuvanted with Matrix-M or AlPO₄. The EBOV/Mak GP specific IgG, IgG1 and IgG2a response, as well as the virus neutralizing antibody response and protection against viral challenge were evaluated.

In summary, our data indicates that co-administration of the Matrix-M adjuvant with purified EBOV/Mak GP nanoparticles provides robust stimulation of the anti-EBOV/Mak GP immune response resulting in 100% protective efficacy in the mouse model. We observed a more rapid onset of anti-EBOV/Mak GP IgG and EBOV neutralization antibodies, increased concentration of IgG2a, as well as increased frequency of multifunctional CD4⁺ and CD8⁺ T cells, T_FH cells, germinal center B cells and persistence of EBOV/Mak GP-specific plasma B cells in the bone marrow. These data, along with the reported safety profile of Matrix-M in clinical trials, have progressed the investigation of this vaccine into NHP studies and a phase I clinical trial. LINK

DARPA-HDIAC Chaperone Proteins

Chaperones are proteins that assist the conformational folding or unfolding of large proteins or macromolecular protein complexes.

HLA-A is a group of human leukocyte antigens (HLA) that are coded for by the HLA-A locus.

HELA cell lines come from Henrietta Lacks and developed at The Johns Hopkins Hospital in Baltimore, Maryland, in 1951. An immortal human CANCEROUS cell line is a cluster of cells that continuously multiply on their own outside of the human from which they originated.

Locus: fixed position [address] on a chromosome where a particular gene or genetic marker is located. In this case it is located at human chromosome 6p21.

HLA is a major histocompatibility complex (MHC) antigen specific to humans. HLA-A is one of three major types of human MHC class I cell surface receptors.

Major histocompatibility complex (MHC) is a large locus on vertebrate DNA containing a set of closely linked polymorphic genes that code for cell surface proteins essential for the adaptive immune system. These cell surface proteins are called MHC molecules.

MHC class I molecules are one of two primary classes of major histocompatibility complex (MHC) molecules (the other being MHC class II) and are found on the cell surface of all nucleated cells in the bodies of vertebrates. They also occur on platelets, but not on red blood cells.

Cell surface receptors (membrane receptors, transmembrane receptors) are receptors that are embedded in the plasma membrane of cells. They act in cell signaling by receiving (binding to) extracellular molecules. They are specialized integral membrane proteins that allow communication between the cell and the extracellular space. The extracellular molecules may be hormones, neurotransmitters, cytokines, growth factors, cell adhesion molecules, or nutrients; they react with the receptor to induce changes in the metabolism and activity of a cell. In the process of signal transduction, ligand binding affects a cascading chemical change through the cell membrane.

The others are HLA-B and HLA-C.

HLA-B (major histocompatibility complex, class I, B) is a human gene that provides instructions for making a protein that plays a critical role in the immune system. HLA-B is part of a family of genes called the human leukocyte antigen (HLA) complex. The HLA complex helps the immune system distinguish the body's own proteins from proteins made by foreign invaders such as viruses and bacteria.

HLA-C belongs to the MHC (human = HLA) class I heavy chain receptors. The C receptor is a heterodimer consisting of a HLA-C mature gene product and β2-microglobulin. The mature C chain is anchored in the membrane. MHC Class I molecules, like HLA-C, are expressed in nearly all cells, and present small peptides to the immune system which surveys for non-self peptides.

The receptor is a heterodimer, and is composed of a heavy α chain and smaller β chain. The α chain is encoded by a variant HLA-A gene, and the β chain (β₂-microglobulin) is an invariant β₂ microglobulin molecule.The β₂ microglobulin protein is coded for by a separate region of the human genome.

Protein Dimer

Hetero Protein Dimer: In biochemistry, a protein dimer is a macromolecular complex formed by two protein monomers, or single proteins, which are usually non-covalently bound. Many macromolecules, such as proteins or nucleic acids, form dimers. The word dimer has roots meaning "two parts", di- + -mer. A protein dimer is a type of protein quaternary structure.

A protein homodimer is formed by two identical proteins. A protein heterodimer is formed by two different proteins.

Most protein dimers in biochemistry are not connected by covalent bonds. An example of a non-covalent heterodimer is the enzyme reverse transcriptase, which is composed of two different amino acid chains. An exception is dimers that are linked by disulfide bridges such as the homodimeric protein NEMO.

Some proteins contain specialized domains to ensure dimerization (dimerization domains) and specificity.

β2 microglobulin also known as B2M is a component of MHC class I molecules, MHC class I molecules have α1, α2, and α3 proteins which are present on all nucleated cells (excludes red blood cells).[5][6] In humans, the β2 microglobulin protein is encoded by the B2M gene.

Once the HLA-A protein is completely translated, it must be folded into the proper shape. A molecular chaperone protein called calnexin and an enzyme called ERp57 assist in the folding process. Calnexin holds the HLA-A heavy chain while Erp57 catalyzes disulfide bonds between the heavy chain and the light, β₂-microglobulin chain. Wikipedia

Chaperone protein: In molecular biology, molecular chaperones are proteins that assist the conformational folding or unfolding [origami] and the assembly or disassembly of other macromolecular structures. Chaperones are present when the macromolecules perform their normal biological functions and have correctly completed the processes of folding and/or assembly. The chaperones are concerned primarily with protein folding.

Calnexin

Calnexin (CNX) is a 67kDa integral protein (that appears variously as a 90kDa, 80kDa, or 75kDa band on western blotting depending on the source of the antibody) of the endoplasmic reticulum (ER). It consists of a large (50 kDa) N-terminal calcium-binding lumenal domain, a single transmembrane helix and a short (90 residues), acidic cytoplasmic tail.

Reading the Neanderthal Code

Smithsonian: LINK Link

They got the human cell line from:

Homo sapiens (human)

Homo sapiens neanderthalensis (Neanderthal)

Homo sapiens subsp. 'Denisova' (Denisova hominin) LINK

German-U.S. collaborative effort embarks on project to sequence the Neanderthal genome

Cite this: Chem. Eng. News 2006, 84, 33, 55

Publication Date:August 14, 2006

https://doi.org/10.1021/cen-v084n033.p055

ANOTHER DAY, ANOTHER GENOME project. But this one is definitely not your average genome project. In fact, the species in question has been extinct for thousands of years.

Last month, Max Planck Institute for Evolutionary Anthropology, in Leipzig, Germany, and 454 Life Sciences, in Branford, Conn., [Provocative name due to book Fahrenheit 454- dystopian society that burns books in order to control dangerous ideas and unhappy concepts, extraordinary suffering and transformation.] announced their plans to sequence the Neanderthal genome.

Among living species, humans share their nearest common ancestor with chimpanzees, which branched off evolutionarily at least 5 million years ago. Neanderthals split off much more recently, somewhere around a half-million years ago. The first Neanderthal fossils were discovered 150 years ago in Germany's Neander Valley.

COVID, Neanderthals & Denisovan

Spans six genes on Chromosome 3.

Genes play a role as well. Last month, researchers compared people in Italy and Spain who became very sick with Covid-19 to those who had only mild infections. They found two places in the genome associated with a greater risk. One is on Chromosome 9 and includes ABO, a gene that determines blood type. The other is the Neanderthal segment on Chromosome 3.

Scientists Covid-19 Host Genetics Initiative released a new set of data downplaying the risk of blood type.

Stronger link between the COVID and the Chromosome 3 segment. People who carry two copies of the variant are three times more likely to suffer from severe illness than people who do not.

Dr. Zeberg decided to find out if the Chromosome 3 segment was passed down from Neanderthals.

Most Neanderthal genes turned out to be harmful to modern humans. They may have been a burden on people’s health or made it harder to have children. As a result, Neanderthal genes became rarer, and many disappeared from our gene pool.

But some genes appear to have provided an evolutionary edge and have become quite common. In May, Max Planck Institute, discovered that one-third of European women have a Neanderthal hormone receptor with increased fertility and fewer miscarriages.

Neanderthal genes that are common today even help us fight viruses. We have held onto those genes ever since.

Chromosome 3 in an online database of Neanderthal genomes.

One version that raises people’s risk of severe Covid-19 is the same version found in a Neanderthal who lived in Croatia 50,000 years ago.

It is possible that an immune response that worked against ancient viruses has ended up overreacting against the new coronavirus. People who develop severe cases of Covid-19 typically do so because their immune systems launch uncontrolled attacks that end up scarring their lungs and causing inflammation.

Dr. Paabo said the DNA segment may account in part for why people of Bangladeshi descent are dying at a high rate of Covid-19 in the United Kingdom.

Neanderthal gene found in many people may open cells to ...

Neanderthal gene found in many people may open cells to coronavirus and increase COVID-19 severity

Neanderthal Genes Could Link to Severe COVID-19

Having Neanderthal genes could be a risk factor for developing severe COVID-19, according to a new study published

Then the story changes that these same genes protect against COVID:

Neanderthal gene protects against COVID-19: Study ...

Neanderthal gene protects against COVID-19 A specific form of a protein passed down from Neanderthals protects against severe

Gene from NEANDERTHALS slashes the risk of dying from Covid-19

People who carry a version of a gene inherited from Neanderthals are at lower risk of serious illness, hospitalization

Neanderthals and Covid-19, beyond the hype | by John Hawks ...

What Neanderthal DNA has to offer is a time dimension. Covid-19 is a new pathogen encountering human biology that goes

DNA Linked to Covid-19 Was Inherited From Neanderthals

A stretch of DNA linked to Covid-19 was passed down from Neanderthals 60,000 years ago, according to a new study.

How Neanderthal DNA affects human health -- including the

Some current-day human DNA still carries traces of the prehistoric sexual interactions early Homo sapiens had with

Neanderthal genes could increase the severity of COVID-19

Neanderthal genes and the impact they could have on COVID-19 patients. How much of the human genome has been inherited

Could COVID-19 have wiped out the Neandertals?

Similar investigations have also identified a protective Neanderthal haplotype on chromosome (chr) 12 that reduces the risk of severe COVID-19, and a protective region on chromosome 9 that is...

Last September announced that the major genetic risk factor for severe COVID-19 is inherited from Neanderthals.

The team found that severe COVID-19 disease is associated with specific genetic variants in six genes within a 50K-base-pair-long region of chromosome 3 that derived directly from a Neanderthal heritage.

Similar investigations have also identified a protective Neanderthal haplotype on chromosome (chr) 12 that reduces the risk of severe COVID-19, and a protective region on chromosome 9 that is associated with the ABO blood groups.

Reports on the bioRxiv preprint server that another exclusively Neandertal variant, this time in the promoter region of the DPP4 gene at chr2q24.2, is really pulling the strings on COVID susceptibility.

DPP4 is a widely expressed extracellular dipeptidyl peptidase involved in immune function and glucose metabolism.

DPP4 is also the receptor gene for the MERS coronavirus.

DPP4 is not a SARS-CoV-2 receptor.

Inhibitors of DPP4 that are already used clinically to treat diabetes appear to have effects on COVID-19 patients.

Immune-associated gene variants including IFNAR2 and TYK2 also control COVID outcomes.

This study also identified DDP9, a sister gene of DD4 residing at chr19p13.3, as a key mediator of inflammatory lung injury.

DPP9 has a similar serine protease activity to DPP4, but differs in that it is not membrane-bound.

The DPP4 gene is not too far away from a long-defunct remnant centromere found nearby in the chr2q21.3–q22.1 region.

There is also an additional vestigial telomere sitting down in the q13 band.

Question of what is it that makes us human, an excellent answer is the fusion of two small ape chromosomes make the human chr2 [via magic].

Do Neandertals have a fused chr2?

Neanderthals have same version of the speech gene, FOXP2.

Human FOXP2, differs from the chimp version in two key places, was famously mutated in the "KE' family from Britain who all had a specific disability in their use of consonants.

In recent COVID risk factor studies:

The problem with this line of work is that we don't have that much sequence data to tell us what makes a Neanderthal a Neanderthal.

Blind Medicine

Blind medicine [prayer] refers to anything done in the absence of personal patient sequence data.

Therefore they want to harvest your blood, so they not be blind, to developing weapons against you. When you submit your blood you just revealed all your secrets and barriers.

Look blind guides in scripture:

Matt. 15:15 Blind guides, ignore them

Matt 23:16

And more Information

MERS DPP4 receptor, no ACE2 receptor variants have emerged as a risk locus for severe COVID-19.

Four variants (rs464397, rs469390, rs2070788 and rs383510) robustly affect expression of the TMPRSS2 serine protease in lung tissue. TMPRSS2-upregulating variants are present at higher frequencies in European and American populations than in the Asian populations.

Antibody-dependent enhancement is said to be dismissed in discussions of COVID. However, recent research now suggests that ADE in COVID is very much a thing.

It is important for the public to get information about the RNA vaccines currently being offered.

Human evolutionary history impacts our COVID-19 risk

The new research into COVID-19 risk and how it intersects with our Neanderthal ancestry is just one more example highlighting viewing

Denisovan DNA influences immune system of modern day ...

Denisovan DNA influences immune system of modern day Oceanian populations

More than 120,000 novel human genetic variations that affect large regions of DNA have been discovered, some of which are linked to immune response, disease susceptibility or digestion. These changes affect multiple bases of DNA, known as structural variations.

These previously-unknown variations in medically-important genes, which could affect the efficacy of medical treatments in certain populations, will be a valuable resource for the field of precision medicine around the globe.

Some genes, such as those that influence immune response, are considered to be 'medically important'. DNA changes affecting how these genes function can lead to health problems or increased resistance or susceptibility to particular diseases.

Scientists at the Wellcome Sanger Institute team then investigated how common these structural variations are in each of the 54 populations, and which of them were inherited from Neanderthal or Denisovan ancestors.

Among the 126,018 structural variations discovered were medically-important variations inherited from Denisovan ancestors in Oceanian populations from Papua New Guinea and nearby, including a high-frequency deletion in the AQR gene that plays a role in detection of viruses and regulation of antiviral immune response.

This is vital knowledge and will help to ensure that treatments can be tailored to each specific population.

The work supports the concept that some human adaptations to different environments are due to the loss or gain of whole genes, or parts of genes.

Key to treating COVID-19 will be found in our DNA | Garvan ...

This TNFAIP3 gene variant traces back to an extinct human species, the Denisovans.

Disease severity is based on our individual immune reaction.

Each of us has six billion letters in our genome and, between any two individuals, there are three million letter differences. We have hundreds of genes essential for our immune system to mount an effective response to pathogens – and a single letter out of six billion can completely throw it off balance. These variants are what make us unique and can also significantly change how we respond to pathogens such as viruses.

Garvan Institute discovered a variant of the gene TNFAIP3, which is common in Indigenous Australians and people throughout Oceania, heightens immune responses.

This TNFAIP3 gene variant traces back to an extinct human species, the Denisovans, and was likely beneficial to survival after modern humans migrated.

Bats, often the origin of virus epidemics, seem to have taken the opposite path: their immune response genes are radically altered in favor of tolerating virus infections. Bats appear to carry and shed the precursors of the coronaviruses that cause SARS, COVID-19 and MERS without it making them sick. Scientists want to biomimicry that response in humans.

Monoclonal antibodies, produced in the lab, could circumvent the genetic variability of people’s own immune responses and be given to provide immediate immunity, both for the treatment of COVID-19 and for prevention, to at-risk individuals, including the elderly, chronically ill and health workers on the frontline.

Neanderthals and Covid-19, beyond the hype | by John Hawks ...

“Viral challenges, bacterial challenges are among the strongest selective forces out there,” says Kelso. Unlike changes in other environmental conditions such as daylight patterns and UV exposure, “pathogens can kill you in one generation.”

Hints of archaic DNA’s role in immune function surfaced as early as 2011, as soon as the Neanderthal genome was available for cross-referencing with sequences from modern humans.

A team led by researchers at Stanford University found that certain human leukocyte antigen (HLA) alleles, key players in pathogen recognition, held signs of archaic ancestry—from Neanderthals, but also from another hominin cousin, the Denisovans.⁶

Several other studies since then have strengthened the link between archaic DNA and immune function, branching out from the HLA system to include numerous other pathways.⁷ For example, multiple labs have tied Neanderthal variants to altered expression levels of genes encoding toll-like receptors (TLRs), key players in innate immune responses.

Toll-like receptors: Toll-like receptors (TLRs) are a class of proteins that play a key role in the innate immune system. They are single-pass membrane-spanning receptors usually expressed on sentinel cells such as macrophages and dendritic cells, that recognize structurally conserved molecules derived from microbes. Once these microbes have breached physical barriers such as the skin or intestinal tract mucosa, they are recognized by TLRs, which activate immune cell responses.

In 2016, Kelso, Dannemann, and a colleague found that pathogen response and susceptibility to develop allergies were associated with Neanderthal sequences that affect TLR production.

Ancient humans procreated with at least four other species.

According to a July 2019 study that describes how our ancestors often mated with other species of the Homo genus: Neanderthals, Denisovans, and two other unnamed hominids.

The discovery was made after scientists used previous studies to create “mixing maps” — aka when and where mating between humans and other hominid species happened. Turns out that these cross-species liaisons happened at times in Europe, and at other times in Asia.

The most-frequent “mixing events” were those between Neanderthals and Homo sapiens.

Neanderthal Traits

Neanderthal DNA influences particular traits in humans.

A combination of archaeological and genetic data also indicates that modern humans mated with Denisovans — another ancient hominid — in eastern Eurasia, maritime South Asia, and later towards Australia. And because East Asians have a second set of Denisovan genomes not found in the DNA of South Asians or people of Papuan descent, scientists believe interbreeding between Denisovans Homo sapiens happened during at least two independent episodes.

Humans also mated with two currently unknown species of ancient hominid, the results suggest. In this study, they are labeled: “EH1” and “EH2.”

EH1 genes have found in the DNA of some modern Asian and Australo-Papuan populations.

EH2, can be found in the genomes of individuals who live near Liang Bua Cave in Flores, Indonesia.

It is currently unclear if these genes are related to Homo floresiensis, an extinct species of hominid whose remains have also been found in Flores. LINK

Smithsonian: LINK Link

They got the human cell line from:

Homo sapiens (human)

Homo sapiens neanderthalensis (Neandertal)

Homo sapiens subsp. 'Denisova' (Denisova hominin) LINK

Matrix M: NVX-CoV2373. AGC Biologics will manufacture Matrix-

Reactions to Novavax!

Adjuvanted COVID-19 Vaccine Caused Severe Adverse Reactions in Clinical Trials LINK

NVX-CoV2373 COVID-19 Vaccine Description

NVX-CoV2373 is a prefusion protein coronavirus vaccine candidate made using Novavax’s proprietary nanoparticle technology, Matrix-M, an adjuvant to enhance immune responses and stimulate high levels of neutralizing antibodies.

LINK

Maryland based Novavax, Inc. is a leading innovator of recombinant vaccines; its proprietary recombinant technology platform combines the power and speed of genetic engineering to efficiently produce highly immunogenic nanoparticles to address urgent global health needs.

AGC Biologics offers deep industry expertise and unique customized services for the scale-up and cGMP manufacture of protein-based therapeutics for commercial mammalian and microbial production.

AGC Biologics Offers:

Plasmid (GMP pDNA) manufacturing,

Cell line development,

Bioprocess development,

Formulation,

Analytical testing,

Antibody drug development and conjugation,

Cell banking and storage and protein expression,

Proprietary CHEF1® Expression System for mammalian production. www.agcbio.com. Another LINK

____________________________________________________________

Celeste Solum is a broadcaster, author, former government, organic farmer and is trained in nursing and environmental medicine. Celeste chronicles the space and earth conditions that trigger the rise and fall of modern & ancient civilizations, calendars, and volatile economies. Cycles are converging, all pointing to a cataclysmic period between 2020 to 2050 in what many scientists believe is an Extinction Level Event.

Tracking goods and people will be a part of managing the population during this convergence.

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